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ON CELLULAR SOURCES FOR INTIMAL HYPERPLASIA AFTER VASCULAR INTERVENTIONS
By Stefan Mellander
[FONT="]Pages:[/FONT][FONT="]49
Publisher:Kompendiet
Edition :2007
Language: English
ISBN: 978-91-628-7203-8
[/FONT][FONT="] ABSTRACT [/FONT][FONT="]
[/FONT]Vascular interventions for the treatment of symptomatic atherosclerosis fail in up to 40% of the cases during the first year. One important reason is the development of a narrowing process known as intimal hyperplasia (IH). The cells forming IH resemble smooth muscle cells (SMCs) from the media of the arterial wall. Therefore the media has generally been regarded as the cellular origin for IH. However, there are reports indicating that other cellular sources might be involved.
The aim of this thesis was to investigate which cellular sources participate in the development of intimal hyperplasia after bypass surgery and balloon injury.
By Stefan Mellander
[FONT="]Pages:[/FONT][FONT="]49
Publisher:Kompendiet
Edition :2007
Language: English
ISBN: 978-91-628-7203-8
[/FONT][FONT="] ABSTRACT [/FONT][FONT="]
[/FONT]Vascular interventions for the treatment of symptomatic atherosclerosis fail in up to 40% of the cases during the first year. One important reason is the development of a narrowing process known as intimal hyperplasia (IH). The cells forming IH resemble smooth muscle cells (SMCs) from the media of the arterial wall. Therefore the media has generally been regarded as the cellular origin for IH. However, there are reports indicating that other cellular sources might be involved.
The aim of this thesis was to investigate which cellular sources participate in the development of intimal hyperplasia after bypass surgery and balloon injury.
[thanks-thanks]pdf,478KB,http://www.mediafire.com/view/?wvtkyfdayajj7rj[/thanks-thanks][FONT="][/FONT]